Tinnitus

The organ of Corti, or spiral organ, is the receptor organ for hearing and is located in the mammalian cochlea. This highly varied strip of epithelial cells allows for transduction of auditory signals into nerve impulses' action potential.[1] Transduction occurs through vibrations of structures in the inner ear causing displacement of cochlear fluid and movement of hair cells at the organ of Corti to produce electrochemical signals. (https://en.wikipedia.org/wiki/Organ_of_Corti)

The external and internal acoustic cells are set with each other with a very sensitive feedback system at the spontaneous oscillation limit. This feedback ensures hearing sensitivity and selectivity very pronounced - on the other hand, the auditory system can easily become unbalanced. If certain acoustic cells no longer work, their neurons are activated by creating an "imaginary" perception in the brain, that is tinnitus. Brain cells in the trunk of the brain or in the time span are constantly overstated. With time they cause a perception of sound even after the pathological stimulus of the organ of Corti has ceased.
Another possibility is that brain cells around those that are no longer stimulated by the Cortian organ begin to get excited by producing the perception of a sound to try to replace brain cells that are no longer stimulated by the organ of Corti.
 
The ciliary cells may alter for the following reasons:
Trauma. An auditory trauma causes a high energy wave in the perilinfa and damages the ciliary cells, especially those located near the oval window and which are responsible for the high sounds.
Intoxications such as mercury, pesticides or other, infections, inflammation, autoimmune diseases, degeneration, age, and more can cause dysfunctions of certain ciliary cells.
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